"I couldn't ask for better equipment," he replied noncommittally. With deft motion of the handler he drew the scalpel down across the chest and along the costal margins in the classic inverted "Y" incision. "We'll take a look at the thorax first," he said, as he used the handlers to pry open the rib cage and expose the thoracic viscera. "Ah! Thought so! See that?" He pointed with a small handler that carried a probe. "Look at those lungs." He swung a viewer into place so Mary could see better. "Look at those abscesses and necrosis. It's Thurston's Disease, all right, with secondary bacterial invasion."

The grayish solidified masses of tissue looked nothing like the normal pink appearance of healthy lungs. Studded with yellowish spherical abscesses they lay swollen and engorged within the gaping cavity of the chest.

"You know the pathogenesis of Thurston's Disease?" Kramer asked.

Mary shook her head, her face yellowish-white in the glare of the fluorescents.

"It begins with a bronchial cough," Kramer said. "The virus attacks the bronchioles first, destroys them, and passes into the deeper tissues of the lungs. As with most virus diseases there is a transitory leukopenia—a drop in the total number of white blood cells—and a rise in temperature of about two or three degrees. As the virus attacks the alveolar structures, the temperature rises and the white blood cell count becomes elevated. The lungs become inflamed and painful. There is a considerable quantity of lymphoid exudate and pleural effusion. Secondary invaders and pus-forming bacteria follow the viral destruction of the lung tissue and form abscesses. Breathing becomes progressively more difficult as more lung tissue is destroyed. Hepatization and necrosis inactivate more lung tissue as the bacteria get in their dirty work, and finally the patient suffocates."

"But what if the bacteria are controlled by antibiotics?"

"Then the virus does the job. It produces atelectasis followed by progressive necrosis of lung tissue with gradual liquefaction of the parenchyma. It's slower, but just as fatal. This fellow was lucky. He apparently stayed out of here until he was almost dead. Probably he's had the disease for about a week. If he'd have come in early, we could have kept him alive for maybe a month. The end, however, would have been the same."

"It's a terrible thing," Mary said faintly.

"You'll get used to it. We get one 

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